Selected Publications

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  • Research Articles
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Host Energy Source Is Important for Disease Tolerance to Malaria.

Tracking Resilience to Infections by Mapping Disease Space.

How Many Parameters Does It Take to Describe Disease Tolerance?

What Can Vampires Teach Us about Immunology?

Defining Resistance and Tolerance to Cancer.

Drosophila melanogaster Natural Variation Affects Growth Dynamics of Infecting Listeria monocytogenes.

The ubiquitin ligase parkin mediates resistance to intracellular pathogens.

Infection-related declines in chill coma recovery and negative geotaxis in Drosophila melanogaster.

How the fly balances its ability to combat different pathogens.

Listeria monocytogenes infection causes metabolic shifts in Drosophila melanogaster.

Disease tolerance as a defense strategy.

Tolerance of Infections.

Reciprocal analysis of Francisella novicida infections of a Drosophila melanogaster model reveal host-pathogen conflicts mediated by reactive oxygen and imd-regulated innate immune response.

The Drosophila TNF ortholog eiger is required in the fat body for a robust immune response.

The role of anorexia in resistance and tolerance to infections in Drosophila.

A signaling protease required for melanization in Drosophila affects resistance and tolerance of infections.

Identification of Drosophila mutants altering defense of and endurance to Listeria monocytogenes infection.

Two ways to survive infection: what resistance and tolerance can teach us about treating infectious diseases.

Drosophila eiger Mutants Are Sensitive to Extracellular Pathogens.

A specific primed immune response in Drosophila is dependent on phagocytes.

Interactions between circadian rhythm and immunity in Drosophila melanogaster.

Akt and FOXO dysregulation contribute to infection-induced wasting in Drosophila.

Secreted Bacterial Effectors and Host-Produced Eiger/TNF Drive Death in a Salmonella-Infected Fruit Fly.

A processed form of the SpƤtzle protein defines dorsal-ventral polarity in the Drosophila embryo.

Dominant and recessive mutations define functional domains of Toll, a transmembrane protein required for dorsal-ventral polarity in the Drosophila embryo.

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